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23) Gerolamo Cardano

Gerolamo Cardano (also Girolamo or Geronimo:  24 September 1501– 21 September 1576) was an Italian polymath whose interests and proficiencies ranged through those of mathematician, physician, biologist, physicist, chemist, astrologer, astronomer, philosopher, music theorist, writer, and gambler. He became one of the most influential mathematicians of the Renaissance and one of the key figures in the foundation of probability; he introduced the binomial coefficients and the binomial theorem in the Western world. He wrote more than 200 works on science.

Cardano partially invented and described several mechanical devices including the combination lock, the gimbal consisting of three concentric rings allowing a supported compass or gyroscope to rotate freely, and the Cardan shaft with universal joints, which allows the transmission of rotary motion at various angles and is used in vehicles to this day. He made significant contributions to hypocycloids - published in De proportionibus, in 1570. The generating circles of these hypocycloids, later named "Cardano circles" or "cardanic circles", were used for the construction of the first high-speed printing presses.

Today, Cardano is well known for his achievements in algebra. In his 1545 book Ars Magna he made the first systematic use of negative numbers in Europe, published (with attribution) the solutions of other mathematicians for cubic and quartic equations, and acknowledged the existence of imaginary numbers.

2404) Cerebral Atrophy

Gist

Cerebral atrophy is the progressive loss of neurons (brain cells) and their connections, resulting in a reduced brain volume or shrinkage. While some atrophy is normal with aging, it can also be a symptom of serious neurological conditions like dementia, stroke, or Huntington's disease, leading to cognitive, motor, and functional impairments. Diagnosis involves imaging tests like MRI, and treatment focuses on managing the underlying condition.

Symptoms of brain atrophy include memory problems, difficulty with problem-solving and decision-making, changes in mood and personality, speech and language difficulties, and impaired motor skills such as coordination and balance. The specific symptoms depend on the affected areas of the brain, and some symptoms like sudden confusion or severe memory loss warrant immediate medical attention.

Summary

Cerebral atrophy is a common feature of many of the diseases that affect the brain. Atrophy of any tissue means a decrement in the size of the cell, which can be due to progressive loss of cytoplasmic proteins. In brain tissue, atrophy describes a loss of neurons and the connections between them. Brain atrophy can be classified into two main categories: generalized and focal atrophy. Generalized atrophy occurs across the entire brain whereas focal atrophy affects cells in a specific location. If the cerebral hemispheres (the two lobes of the brain that form the cerebrum) are affected, conscious thought and voluntary processes may be impaired.

Some degree of cerebral shrinkage occurs naturally with the dynamic process of aging. Structural changes continue during adulthood as brain shrinkage commences after the age of 35, at a rate of 0.2% per year. The rate of decline is accelerated when individuals reach 70 years old. By the age of 90, the human brain will have experienced a 15% loss of its initial peak weight. Besides brain atrophy, aging has also been associated with cerebral microbleeds.

Details

Brain atrophy refers to a loss of brain cells, or a loss in the number of connections between brain cells. It can occur as a result of the natural aging process, injury, infection, or certain health conditions.

There are two main types of brain atrophy: focal atrophy, which occurs in specific brain regions, and generalized atrophy, which occurs across the brain.

People who experience brain atrophy typically develop lower cognitive functioning as a result of this type of brain damage.

This article describes the symptoms and causes of brain atrophy.

Symptoms of brain atrophy

The symptoms of brain atrophy will vary depending on the location of the atrophy and its severity. They may include:

Seizures

A seizure is a sudden spike of electrical activity in the brain. There are two main types of seizure: partial seizures, which affects just one part of the brain, and generalized seizures, which affects both sides of the brain.

The symptoms of a seizure depend on which part of the brain it affects. Some people may not experience any noticeable symptoms, whereas others may experience one or more of the following:

* behavioral changes
* jerking eye movements
* a bitter or metallic taste in the mouth
* drooling or frothing at the mouth
* teeth clenching
* grunting and snorting
* muscle spasms
* convulsions
* loss of consciousness

Aphasia

Aphasia refers to a group of symptoms that affect a person’s ability to communicate. Some types of aphasia can affect a person’s ability to produce or understand speech. Others can affect a person’s ability to read or write.

According to the National Aphasia Association, there are eight different types of aphasia. The type of aphasia a person experiences depends on the part or parts of the brain that sustain damage.

Some cases of aphasia are relatively mild, whereas others may severely impair a person’s ability to communicate.

Dementia

Dementia refers to a continuing decline in brain function. The symptoms may include:

* memory loss
* difficulty with reasoning or judgment
* difficulty with language or communication
* problems with movement and coordination
* mood or personality changes
* hallucinations
* difficulty carrying out daily activities

There are several different types of dementia. Alzheimer’s disease is the most common.

A person’s risk of dementia increases with age. However, it is not a natural part of the aging process.

Causes of brain atrophy

Brain atrophy can occur as a result of injury, either from a traumatic brain injury (TBI) or a stroke. It may also occur as a result of one of infections, such as HIV or those that cause brain inflammation (encephalitis).

In some cases, brain atrophy may occur as a result of a chronic condition, such as:

* multiple sclerosis (MS)
* Huntington’s disease
* Alzheimer’s disease
* Parkinson’s disease
* cerebral palsy
* leukodystrophies, which are a group of rare genetic conditions affecting the nervous system

Diagnosis

When diagnosing brain atrophy, a doctor may begin by taking a full medical history and asking about a person’s symptoms. This may include asking questions about when the symptoms began and if there was an event that triggered them.

The doctor may also carry out language or memory tests or other specific tests of brain function.

If they suspect that a person has brain atrophy, they will need to locate the brain damage and assess its severity. This will require an MRI or CT scan.

Additional Information

Brain atrophy (cerebral atrophy) is a loss of neurons and connections between neurons. Different conditions cause brain atrophy, including cerebral palsy, dementia and infectious diseases. Symptoms and severity of brain atrophy depend on the specific disease and location of damage. Treatment involves managing the underlying disorder.

What is brain atrophy?

People with brain atrophy, also called cerebral atrophy, lose brain cells (neurons), and connections between their brain cells and brain volume often decreases. This loss can lead to problems with thinking, memory and performing everyday tasks. The greater the loss, the more impairment someone has.

There are two types of brain atrophy:

* Focal: Damage occurs in one area of your brain.
* Generalized: Damage expands to your entire brain.

Is brain atrophy a normal part of aging?

People lose some brain cells as they get older, and brain volume decreases as well, but healthcare providers use the term “brain atrophy” when a person has more brain changes than expected for age. Here, the damage happens faster than the typical aging process.

Who is at risk for brain atrophy?

Some factors may increase your chances of developing brain atrophy, such as:

* Advanced age.
* Family history of genetic disorders, such as Huntington’s disease.
* Family history of neurological disorders, such as Alzheimer’s disease.
* Head or brain injury.
* Heavy drinking (alcohol use disorder).
* Smoking.

Does brain atrophy lead to dementia?

There’s a connection between brain atrophy and dementia. Specifically, dementia causes extreme brain atrophy. Dementia is a general term that describes severe thinking problems that interfere with daily life.

The most common type of dementia is Alzheimer’s disease.

Does brain atrophy cause aphasia?

People with aphasia (speaking and language problems) as part of an underlying neurodegenerative disease like Alzheimer’s disease often have brain atrophy as well. Here, damage occurs in areas responsible for producing and processing language. This disorder ranges in severity. Some people have trouble recalling the correct name for people, places and things. Others are completely unable to communicate.

Symptoms and Causes:

What causes brain atrophy?

Many different disorders, neurodegenerative diseases, infectious diseases and severe injuries can cause brain atrophy, including:

* Cerebral palsy.
* Encephalitis.
* HIV and AIDS.
* Huntington’s disease.
* Leukodystrophies.
* Multiple sclerosis.
* Stroke.
* Syphilis.
* Traumatic brain injury.
* Alzheimer’s disease.

What are the symptoms of brain atrophy?

Symptoms of brain atrophy vary depending on which specific part of your brain is damaged. Symptoms also range from mild to severe.

In general, brain atrophy happens with various conditions, and symptoms can vary to include:

Aphasia

* Difficulty speaking.
* Difficulty writing.
* Inability to understand the meaning of words.

Dementia

* Hallucinations.
* Loss of language.
* Memory problems.
* Mood and personality changes.
* Poor judgment.

Seizures

* Bitter or metallic taste.
* Convulsions.
* Loss of consciousness.
* Spasms.
* Teeth clenching.

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#5781. What does the noun cliché mean?

#5782. What does the noun clientele mean?

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See the links: Introduction to Calculus.

2351) Konrad Lorenz

Gist:

Work

Some animal and human patterns of behavior are innate. Examples of such behavioral patterns in animals can be seen in how they convey information to one another, how they behave when mating and how they care for their young. Karl von Frisch, Konrad Lorenz and Nikolaas Tinbergen made pioneering contributions within ethnology by studying animal behavior. Among other things, Konrad Lorenz revealed in the 1930s that birds hatched in an incubator without the presence of their parents follow whatever they first catch sight of. For example, they can become fixated on a person.

Summary

Konrad Lorenz (born Nov. 7, 1903, Vienna, Austria—died Feb. 27, 1989, Altenburg) was an Austrian zoologist and the founder of modern ethology, the study of animal behaviour by means of comparative zoological methods. His ideas contributed to an understanding of how behavioral patterns may be traced to an evolutionary past, and he was also known for his work on the roots of aggression. He shared the Nobel Prize for Physiology or Medicine in 1973 with the animal behaviourists Karl von Frisch and Nikolaas Tinbergen.

Lorenz was the son of an orthopedic surgeon. He showed an interest in animals at an early age, and he kept animals of various species—fish, birds, monkeys, dogs, cats, and rabbits—many of which he brought home from his boyhood excursions. While still young, he provided nursing care for sick animals from the nearby Schönbrunner Zoo. He also kept detailed records of bird behaviour in the form of diaries.

In 1922, after graduating from secondary school, he followed his father’s wishes that he study medicine and spent two semesters at Columbia University, in New York City. He then returned to Vienna to study.

During his medical studies Lorenz continued to make detailed observations of animal behaviour; a diary about a jackdaw that he kept was published in 1927 in the prestigious Journal für Ornithologie. He received an M.D. degree at the University of Vienna in 1928 and was awarded a Ph.D. in zoology in 1933. Encouraged by the positive response to his scientific work, Lorenz established colonies of birds, such as the jackdaw and greylag goose, published a series of research papers on his observations of them, and soon gained an international reputation.

In 1935 Lorenz described learning behaviour in young ducklings and goslings. He observed that at a certain critical stage soon after hatching, they learn to follow real or foster parents. The process, which is called imprinting, involves visual and auditory stimuli from the parent object; these elicit a following response in the young that affects their subsequent adult behaviour. Lorenz demonstrated the phenomenon by appearing before newly hatched mallard ducklings and imitating a mother duck’s quacking sounds, upon which the young birds regarded him as their mother and followed him accordingly.

In 1936 the German Society for Animal Psychology was founded. The following year Lorenz became coeditor in chief of the new Zeitschrift für Tierpsychologie, which became a leading journal for ethology. Also in 1937, he was appointed lecturer in comparative anatomy and animal psychology at the University of Vienna. From 1940 to 1942 he was professor and head of the department of general psychology at the Albertus University at Königsberg, Germany (now Kaliningrad, Russia).

From 1942 to 1944 he served as a physician in the German army and was captured as a prisoner of war in the Soviet Union. He was returned to Austria in 1948 and headed the Institute of Comparative Ethology at Altenberg from 1949 to 1951. In 1950 he established a comparative ethology department in the Max Planck Institute of Buldern, Westphalia, becoming codirector of the Institute in 1954. From 1961 to 1973 he served as director of the Max Planck Institute for Behaviour Physiology, in Seewiesen. In 1973 Lorenz, together with Frisch and Tinbergen, was awarded the Nobel Prize for Physiology or Medicine for their discoveries concerning animal behavioral patterns. In the same year, Lorenz became director of the department of animal sociology at the Institute for Comparative Ethology of the Austrian Academy of Sciences in Altenberg.

Lorenz’s early scientific contributions dealt with the nature of instinctive behavioral acts, particularly how such acts come about and the source of nervous energy for their performance. He also investigated how behaviour may result from two or more basic drives that are activated simultaneously in an animal. Working with Nikolaas Tinbergen of the Netherlands, Lorenz showed that different forms of behaviour are harmonized in a single action sequence.

Lorenz’s concepts advanced the modern scientific understanding of how behavioral patterns evolve in a species, particularly with respect to the role played by ecological factors and the adaptive value of behaviour for species survival. He proposed that animal species are genetically constructed so as to learn specific kinds of information that are important for the survival of the species. His ideas have also cast light on how behavioral patterns develop and mature during the life of an individual organism.

In the latter part of his career, Lorenz applied his ideas to the behaviour of humans as members of a social species, an application with controversial philosophical and sociological implications. In a popular book, Das sogenannte Böse (1963; On Aggression), he argued that fighting and warlike behaviour in man have an inborn basis but can be environmentally modified by the proper understanding and provision for the basic instinctual needs of human beings. Fighting in lower animals has a positive survival function, he observed, such as the dispersion of competitors and the maintenance of territory. Warlike tendencies in humans may likewise be ritualized into socially useful behaviour patterns. In another work, Die Rückseite des Spiegels: Versuch einer Naturgeschichte menschlichen Erkennens (1973; Behind the Mirror: A Search for a Natural History of Human Knowledge), Lorenz examined the nature of human thought and intelligence and attributed the problems of modern civilization largely to the limitations his study revealed.

Details

Konrad Zacharias Lorenz (7 November 1903 – 27 February 1989) was an Austrian zoologist, ethologist, and ornithologist. He shared the 1973 Nobel Prize in Physiology or Medicine with Nikolaas Tinbergen and Karl von Frisch. He is often regarded as one of the founders of modern ethology, the study of animal behavior. He developed an approach that began with an earlier generation, including his teacher Oskar Heinroth.

Lorenz studied instinctive behavior in animals, especially in greylag geese and jackdaws. Working with geese, he investigated the principle of imprinting, the process by which some nidifugous birds (i.e. birds that leave their nest early) bond instinctively with the first moving object that they see within the first hours of hatching. Although Lorenz did not discover the topic, he became widely known for his descriptions of imprinting as an instinctive bond. In 1936, he met Tinbergen, and the two collaborated in developing ethology as a separate sub-discipline of biology. A Review of General Psychology survey, published in 2002, ranked Lorenz the 65th most cited scholar of the 20th century in the technical psychology journals, introductory psychology textbooks, and survey responses.

Lorenz's work was interrupted by the onset of World War II and in 1941 he was recruited into the German Army as a medic. In 1944, he was sent to the Eastern Front where he was captured by the Soviet Red Army and spent four years as a German prisoner of war in Soviet Armenia. After the war, he regretted his membership in the Nazi Party.

Lorenz wrote numerous books, some of which, such as King Solomon's Ring, On Aggression, and Man Meets Dog, became popular reading. His last work Here I Am – Where Are You? is a summary of his life's work and focuses on his famous studies of greylag geese.

Biography

Lorenz was the son of Adolf Lorenz, a wealthy and distinguished surgeon, and his wife Emma (née Lecher), a physician who had been her husband's assistant. The family lived on a large estate at Altenberg, and had a city apartment in Vienna. He was educated at the Public Schottengymnasium of the Benedictine monks in Vienna.

In his autobiographical essay, published in 1973 in Les Prix Nobel (winners of the prizes are requested to provide such essays), Lorenz credits his career to his parents, who "were supremely tolerant of my inordinate love for animals", and to his childhood encounter with Selma Lagerlöf's The Wonderful Adventures of Nils, which filled him with a great enthusiasm about wild geese."

At the request of his father, Adolf Lorenz, he began a premedical curriculum in 1922 at Columbia University, but he returned to Vienna in 1923 to continue his studies at the University of Vienna. He graduated as Doctor of Medicine (MD) in 1928 and became an assistant professor at the Institute of Anatomy until 1935. He finished his zoological studies in 1933 and received his second doctorate (PhD).

While still a student, Lorenz began developing what would become a large menagerie, ranging from domestic to exotic animals. In his popular book King Solomon's Ring, Lorenz recounts that while studying at the University of Vienna he kept a variety of animals at his parents' apartment, ranging from fish to a capuchin monkey named Gloria.

In 1936, at an international scientific symposium on instinct, Lorenz met his great friend and colleague Nikolaas Tinbergen. Together they studied geese—wild, domestic, and hybrid. One result of these studies was that Lorenz "realized that an overpowering increase in the drives of feeding as well as of copulation and a waning of more differentiated social instincts is characteristic of very many domestic animals". Lorenz began to suspect and fear "that analogous processes of deterioration may be at work with civilized humanity." This observation of bird hybrids caused Lorenz to believe that domestication resulting from urbanisation in humans might also cause dysgenic effects, and to argue in two papers that the Nazi eugenics policies against this were therefore scientifically justified.

In 1940 he became a professor of psychology at the University of Königsberg. He was drafted into the Wehrmacht in 1941. He sought to be a motorcycle mechanic, but instead he was assigned as a military psychologist, conducting racial studies on humans in occupied Poznań under Rudolf Hippius. The objective was to study the biological characteristics of "German-Polish half-breeds" to determine whether they 'benefited' from the same work ethics as 'pure' Germans. The degree to which Lorenz participated in the project is unknown, but the project director Hippius referred a couple of times to Lorenz as an "examining psychologist".

Lorenz later described that he once saw transports of concentration camp inmates at Fort VII near Poznań, which made him "fully realize the complete inhumanity of the Nazis".

He was sent to the Russian front in 1944 where he quickly became a prisoner of war in the Soviet Union from 1944 to 1948. In captivity in Soviet Armenia, he continued to work as a medic and "became tolerably fluent in Russian and got quite friendly with some Russians, mostly doctors." When he was repatriated, he was allowed to keep the manuscript of a book he had been writing and his pet starling. He arrived back in Altenberg (his family home, near Vienna) both "with manuscript and bird intact." The manuscript became his 1973 book Behind the Mirror.

The Max Planck Society established the Lorenz Institute for Behavioral Physiology in Buldern, Germany, in 1950. In his memoirs, Lorenz described the chronology of his war years differently from what historians have been able to document after his death. He himself claimed that he was captured in 1942, where in reality he was only sent to the front and captured in 1944, leaving out entirely his involvement with the Poznań project.

In 1958, Lorenz transferred to the Max Planck Institute for Behavioral Physiology in Seewiesen. He shared the 1973 Nobel Prize in Physiology or Medicine "for discoveries in individual and social behavior patterns" with two other important early ethologists, Nikolaas Tinbergen and Karl von Frisch. In 1969, he became the first recipient of the Prix mondial Cino Del Duca. He was a friend and student of renowned biologist Sir Julian Huxley (grandson of "Darwin's bulldog", Thomas Henry Huxley). Famed psychoanalyst Ralph Greenson and Sir Peter Scott were good friends. Lorenz and Karl Popper were childhood friends; many years after they met, during the celebration of Popper's 80 years, they wrote together a book entitled Die Zukunft ist offen.

He retired from the Max Planck Institute in 1973 but continued to research and publish from Altenberg and Grünau im Almtal in Austria. He died on 27 February 1989 in Altenberg.

Personal life

Lorenz wed his childhood companion, Margarethe Gebhardt, a gynaecologist and the daughter of a local market gardener.; the couple had three children, one son and two daughters. He resided on the Lorenz family estate, notable for its "fantastical neo-baroque mansion", formally belonging to his father.

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#5779. What does the noun doorway mean?

#5780. What does the noun dopiaza mean?

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#9749.